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GH accelerates oncogenic signaling for common oncogenic processes and tumor development, including proliferation, migration, invasion (Brunet-Dunand et al., 2009), metastasis (Brittain et al., 2017), and epithelial-to-mesenchymal transition (EMT) (Mukhina et al., 2004), and prevents apoptosis by exerting autocrine or paracrine effects on cancer cell behavior and neighboring cells within the tumor microenvironments (Perry et al., 2017)

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As chemical biology, nanomedicine and mitochondrial genetics continue to intersect, mitochondrial targeting is shifting from a delivery solution to a conceptual foundation for neurodegenerative therapy (Figure 6D)